Clinical Heterogeneity in two patients with Noonan-like Syndrome associated with the same SHOC2 mutation

نویسندگان

  • Donatella Capalbo
  • Maria Giuseppa Scala
  • Daniela Melis
  • Giorgia Minopoli
  • Nicola Improda
  • Loredana Palamaro
  • Claudio Pignata
  • Mariacarolina Salerno
چکیده

Noonan-like syndrome with loose anagen hair (NS/LAH; OMIM #607721) has been recently related to the invariant c.4A > G missense change in SHOC2. It is characterized by features reminiscent of Noonan syndrome. Ectodermal involvement, short stature associated to growth hormone (GH) deficiency (GHD), and cognitive deficits are common features. We compare in two patients with molecularly confirmed NS/LAH diagnosis, the clinical phenotype and pathogenetic mechanism underlying short stature. In particular, while both the patients exhibited a severe short stature, GH/IGFI axis functional evaluation revealed a different pathogenetic alteration, suggesting in one patient an upstream alteration (typical GHD) and in the other one a peripheral GH insensitivity.

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Noonan syndrome is a genetic condition inherited by an autosomally dominant manner, characterised by congenital heart disease, short stature, abnormal facies and the somatic feature of Turner's syndrome, but a normal karyotype. Noonan syndrome affects approximately 1 in 1500 live births. Congenital heart disease occurs in 35-50% of patients diagnosed with noonan syndrome. The most common cardia...

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عنوان ژورنال:

دوره 38  شماره 

صفحات  -

تاریخ انتشار 2012